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This review will provide an educational update of the scientific evidence for the efficacy and adverse effects of NSAIDs in view of the emerging new information for this class of drugs.
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In these patients, administration of a NSAID may cause a dose-dependent reduction in prostaglandin formation and, secondarily, in renal blood flow, which may precipitate overt renal decompensation.
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Knowledge of risk factors for oral and craniofacial diseases and disorders allows other health care providers to screen for these risk factors and contribute to improving oral health.
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Patients receiving Ketorolac tromethamine who may be adversely affected by alterations in platelet function, such as those with coagulation disorders or patients receiving anticoagulants, should be carefully monitored.
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The recommended total daily dose of ketorolac tromethamine tablets (maximum 40 mg) is significantly lower than for ketorolac tromethamine injection (maximum 120 mg).
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In the absence of similar close attention to clinical and pathologic details, the number of deaths causally attributed to acetaminophen, because of its incidental use in a large percentage of the population (23% in the past week), may be grossly exaggerated.
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The purpose of this systematic review is to assess the effectiveness of ketorolac vs control in the treatment of acute pseudophakic CME (necessitating therapeutic intervention within 4 months of CME onset).
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Implemented McNeil initiatives include consumer and healthcare educational programs and an ongoing consumer OTC analgesic use monitoring survey.
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