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In these patients, administration of a NSAID may cause a dose-dependent reduction in prostaglandin formation and, secondarily, in renal blood flow, which may precipitate overt renal decompensation.

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Knowledge of risk factors for oral and craniofacial diseases and disorders allows other health care providers to screen for these risk factors and contribute to improving oral health.

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Patients receiving Ketorolac tromethamine who may be adversely affected by alterations in platelet function, such as those with coagulation disorders or patients receiving anticoagulants, should be carefully monitored.

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The recommended total daily dose of ketorolac tromethamine tablets (maximum 40 mg) is significantly lower than for ketorolac tromethamine injection (maximum 120 mg).

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In the absence of similar close attention to clinical and pathologic details, the number of deaths causally attributed to acetaminophen, because of its incidental use in a large percentage of the population (23% in the past week), may be grossly exaggerated.

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It can fail to reveal some unusual radiolucent stones, such as those caused by indinavir and atazanavir, which are typically invisible on the CT scan (though some serve as a nidus for deposition of calcium oxalate or calcium phosphate deposition and thus become radiopaque).

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